Abstracts: Virulence Determinants
Virulence Determinants
- Bacteriophages facilitate nasal colonization of Staphylococcus aureus
- Characterization a novel DNA-binding protein that modulates β-lactam resistance in Staphylococcus aureus
- Characterization of virulence gene expression in different Staphylococcus aureus mastitis isolates
- Examining specific virulence factors involved in Staphylococcus aureus keratitis
- Genetic background of Staphylococcus aureus strains isolated in Lebanon
- Genotype variability of the enterotoxin H-positive Staphylococcus aureus strains isolated from patients and foods in the Czech Republic
- High Panton-Valentine leukocidin prevalence among community-associated methicillin-sensitive Staphylococcus aureus from Indonesia
- Identification and characterization of rsr, a novel inhibitor of Staphylococcus aureus virulence
- Molecular characterization of the Staphylococcus aureus surface protein C (SasC) involved in cell aggregation and biofilm formation
- New SEB-encoding Staphylococcus aureus pathogenicity island
- Panton-Valentine Leukocidin (PVL)-positive and -negative community-associated MRSA (CA-MRSA) USA400 sibling strains exhibit similar virulence characteristics in in vitro cell Cclture and animal models
- Prevalence of Panton-Valentine Leukocidin (PVL) in Staphylococcus aureus isolates causing bacteraemia from a paediatric/obstetric hospital in South Australia 2004 - 2008
- Proteolytic regulation of antitoxins in S. aureus by ClpCP
- PVL-positivity among clinical and colonizing isolates of Staphylococcus aureus from children in south-west of Sweden
- Staphylococcal enterotoxin A induces emesis through increasing serotonin release in intestine and endogenous cannabinoid system protects against the emetic response
- Staphylococcal superantigen-like protein 10: Targeting of host immune defences by Staphylococcus aureus
- Staphylococcal superantigen-like protein 14 inhibits the human complement cascade
- Staphylococcal superantigen-like protein 9 (SSL9): a novel complement regulator of S. aureus
- Staphylococcal virulence and pathogenesis: Why so many toxins?
- T cell activation by enterotoxin gene cluster-encoded and ’classical’ Staphylococcus aureus superantigens
- The virulence gene expression pattern of different clinical isolates of Staphylococcus aureus is more heterogeneous than expected from the genomic situation
21 abstracts